The RADIL has a primary research mission of investigating diseases in research animals. Significant outcomes of these disease studies include identification and characterization of newly-recognized disease agents, description of animal models of human disease and development of state-of-the-art diagnostic tests to identify rodent disease agents that would adversely affect animal-based research. The combined activiities of the RADIL research and diagnostic laboratories naturally provide an enriched environment for training of veterinary residents and graduate students.
11.19.2009
In the past, in order to accurately determine the presence of pinworms in an animal has required a post-mortem direct exam. With the introduction of RADIL's new PCR assay which tests for both Syphacia obvelata and Aspiculuris tetraptera, antemortem testing can now be performed with highly accurate results. RADIL's Pinworm PCR assay is nearly as sensitive as the direct exam and has the advantage that the animal does not need to be euthanized for evaluation. In studies, it was also more sensitive than either of the two antemortem tests (tape test and fecal float).
Pinworm by PCR evaluation will be available beginning December 1, 2009 as part of the Mouse Basic , Mouse Comprehensive and Rat Basic Fecal Panels, as a Helicobacter & Pinworm panel, or as a stand-alone assay. For more information and pricing, please click here.
( for more info click here )11.19.2009
At this year's National AALAS Meeting in Denver, Colorado, RADIL 
introduced a breakthrough serologic testing technology that will offer clients an increased level of results confidence for the most prevalent mouse and rat agents. MFI2 represents an advanced approach to serologic monitoring for laboratory animal pathogens, providing the highest level of diagnostic accuracy available. By evaluating multiple antigens for each agent, primary and confirmatory testing now occur at the same time, saving time and increasing the predictive value of the final results. Clients will begin seeing multiple antigens reported on case reports as of December 1, 2009.
For more information regarding MFI2, please visit the Serology section of this site.
( for more info click here )Tippens AS, Davis SV, Hayes JR, Bryda EC, Green TL, Gruetter CA. Detection of histidine decarboxylase in rat aorta and cultured rat aortic smooth muscle cells. Inflamm Res. 2004 Aug;53(8):390-5. Epub 2004 Aug 10.
Walker E, Black J, Parris C, Bryda EC, Cansino S, Hunt L, Chappell J, Wehner P, Studeny M, Wright GL. Effect of experimental hyperhomocysteinemia on cardiac structure and function in the rat. Ann Clin Lab Sci. 2004 Spring;34(2):175-80
Cogswell C, Price SJ, Hou X, Guay-Woodford LM, Flaherty L, Bryda EC. Positional cloning of jcpk/bpk locus of the mouse. Mamm Genome. 2003 Apr;14(4):242-9.
Price SJ, Chittenden LR, Flaherty L, O'Dell B, Guay-Woodford LM, Stubbs L, Bryda EC. Characterization of the region containing the jcpk PKD gene on mouse Chromosome 10. Cytogenet Genome Res. 2002;98(1):61-6.
Chittenden L, Lu X, Cacheiro NL, Cain KT, Generoso W, Bryda EC, Stubbs L. A new mouse model for autosomal recessive polycystic kidney disease. Genomics. 2002 Apr;79(4):499-504.
Bryda EC, Kim HJ, Legare ME, Frankel WN, Noben-Trauth K. High-resolution genetic and physical mapping of modifier-of-deafwaddler (mdfw) and Waltzer (Cdh23v). Genomics. 2001 May 1;73(3):338-42.
Bolz H, von Brederlow B, Ramirez A, Bryda EC, Kutsche K, Nothwang HG, Seeliger M, del C-Salcedo Cabrera M, Vila MC, Molina OP, Gal A, Kubisch C. Mutation of CDH23, encoding a new member of the cadherin gene family, causes Usher syndrome type 1D. Nat Genet. 2001 Jan;27(1):108-12.
Di Palma F, Holme RH, Bryda EC, Belyantseva IA, Pellegrino R, Kachar B, Steel KP, Noben-Trauth K. Mutations in Cdh23, encoding a new type of cadherin, cause stereocilia disorganization in waltzer, the mouse model for Usher syndrome type 1D. Nat Genet. 2001 Jan;27(1):103-7.
©2007 - Curators of the University of Missouri. All rights reserved. DMCA and other copyright information.
An equal opportunity/affirmative action institution.
design provided by Nine Plus One